Reduced renal blood flow and glomerular filtration rate are the hallmarks of hepatorenal syndrome (HRS), the manifestation of renal dysfunction in cirrhotic patients. When kidney function tests are deranged, yet there is no sign of intrinsic kidney disease, such as proteinuria, hematuria, or abnormal kidney ultrasonography, the condition is known as hepatorenal syndrome. Hepatorenal syndrome, which results from functional alterations in the renal circulation and is treatable with vasoconstrictor medications, is different from other causes of acute kidney injury (AKI). Depending on how severe and rapidly the kidney injury is progressing, there are two distinct types of hepatorenal syndrome. The first, HRS-AKI, is an acute kidney dysfunction, whereas the second, HRS-chronic kidney disease (CKD), is a more persistent kidney dysfunction. In this review, we discuss the definition, pathophysiology, assessment, management, and future perspective of renal dysfunction in cirrhosis.