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After exposure to pollutants, their transgenerational translocation could be formed in Caenorhabditis elegans. Transgenerational translocation of particle pollutants may be somewhat different from transgenerational translocation of organic pollutants. ACS-22, CLK-1, ISP-1, and DAF-2 were identified as important regulators for the control of transgenerational translocation of pollutants. Transgenerational damage on intestinal barrier function could be induced by exposure to particle and organic pollutants. ACS-22, ERM-1, HMP-2, PKC-3, and immunity response proteins (such as LYS-7 and SPP-1) functioned to regulate the transgenerational damage on intestinal barrier function by pollutants.

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Roles of Transgenerational Translocation and Biological Barrier in Controlling Transgenerational Toxicity of Pollutants

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  • 发表日期：

    2026年

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  • Dayong Wang

摘要

After exposure to pollutants, their transgenerational translocation could be formed in Caenorhabditis elegans. Transgenerational translocation of particle pollutants may be somewhat different from transgenerational translocation of organic pollutants. ACS-22, CLK-1, ISP-1, and DAF-2 were identified as important regulators for the control of transgenerational translocation of pollutants. Transgenerational damage on intestinal barrier function could be induced by exposure to particle and organic pollutants. ACS-22, ERM-1, HMP-2, PKC-3, and immunity response proteins (such as LYS-7 and SPP-1) functioned to regulate the transgenerational damage on intestinal barrier function by pollutants.