The internal nasal valve is an anatomically dynamic and complex area of the nasal cavity. This area is the narrowest region in the upper airway that can account for up to 70% of nasal respiratory resistance [1]. Anatomically, it is composed of the dorsal septum, upper lateral cartilage, and head of the inferior turbinate (Fig. 19.1). Both static (fixed) and dynamic (motion) challenges lead to nasal airway resistance. Static abnormalities include deviated nasal septum, inferior turbinate hypertrophy, and malpositioned (narrow) upper lateral cartilage. Dynamic abnormalities impact nasal resistance during active nasal inspiration when the sidewalls of the nose collapse due to weak upper lateral cartilage. This condition highlights Bernoulli’s principle describing that as fluid velocity increases in a tube due to narrowing, the pressure in that tube must decrease. This decrease in pressure is akin to a vacuum effect and pulls in the surrounding inadequately supported tissue of the lateral nasal wall. The pathology of this event is called Nasal Valve Compromise (NVC), also called nasal valve collapse, and primarily relates to the internal nasal valve [2].