Raynaud’s syndrome is a vascular disorder characterized by episodic vasospasm of the small arteries and arterioles, usually in the fingers and toes, in response to cold temperatures or emotional stress. These spasms cause a distinctive color change in the skin—typically white (pallor), then blue (cyanosis), and finally red (rubor) as blood flow returns. There are two main types: Primary Raynaud’s, which occurs without an associated medical condition and is often idiopathic vasospastic condition, and Secondary Raynaud’s, which is linked to underlying autoimmune or non-autoimmune diseases which cause obstruction and destruction of digital arteries and capillaries such as systemic sclerosis. Secondary Raynaud’s tends to be more severe and may lead to complications like ulcers or tissue damage. Symptoms typically include numbness, tingling, and pain during attacks, followed by throbbing or swelling when circulation returns. Episodes can last from minutes to hours and vary in frequency and intensity. Diagnosis is largely clinical, based on history and physical examination. Nailfold capillaroscopy and blood tests (e.g. cryoglobulinaemia assay, serum protein electrophoresis/immunoelectrophoresis, thyroid hormones, and antinuclear-antibodies ANA test) may help identify secondary causes. Treatment focuses on avoiding triggers like cold and stress, and in severe cases, medications such as calcium channel blockers (e.g. nifedipine long acting) or vasodilators may be prescribed. In extreme cases, surgical interventions like sympathectomy can be considered. Raynaud’s syndrome affects quality of life, but is generally manageable with appropriate care and preventive strategies. Lifestyle modifications, including warming techniques and smoking cessation, are essential in managing the condition. While primary Raynaud’s is manageable, secondary Raynaud’s requires monitoring and treatment of the underlying disease.