Abstract <p><b>Objective:</b> There is increasing evidence that excessive exposure to copper sulfate (Cu<sup>2+</sup>) can cause detrimental effects on various human organs, with a notable concentration of this substance found in the kidneys. This study was to evaluate the impact of intraperitoneal administration of Cu<sup>2+</sup> at a dose of 10 mg/kg and investigate the protective effects of Vitamin C, administered at 80 and 100 mg/kg, on the kidneys of male Wistar rats for 10 days, with injections given once a day for one minute. <b>Materials and Methods:</b> Changes in renal histological, stereological, and biochemical parameters were evaluated. <b>Results:</b> The intraperitoneal administration of Cu<sup>2+</sup> at 10 mg/kg, unlike Vitamin C (80 and 100 mg/kg), resulted in significant histological and stereological changes in the kidneys of male Wistar rats. These changes included vascular hyperemia, extensive hemorrhage, tissue atrophy, and a reduction in the volume of the left kidney (<i>p </i>&lt; 0.05). Furthermore, exposure to Cu<sup>2+</sup> resulted in oxidative stress, as indicated by a reduction in SOD activity and an elevation in MDA levels in the right kidney (<i>p</i> &lt; 0.05). Notably, the higher dose of Vitamin C mitigated the adverse effects of Cu<sup>2+</sup> on the histological, stereological, and biochemical parameters compared to the Cu<sup>2+</sup>-only group (<i>p </i>&lt; 0.05). <b>Conclusion:</b> Our results proposed that co-treatment with Vitamin C may reverse Cu<sup>2+</sup>-induced alterations and provide a significant protective effect.</p>

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Examining the Histological and Stereological Alterations, Along with Oxidative Stress Markers, in the Kidney Tissues of Male Rats Subjected to Copper Sulfate Toxicity and Treated with Ascorbic Acid

  • Bita Delir,
  • Homiera Hatami Nemati,
  • Hatam Ahmadi,
  • Seyed Mahdi Banan Khojasteh,
  • Gholamreza Hamidian

摘要

Abstract

Objective: There is increasing evidence that excessive exposure to copper sulfate (Cu2+) can cause detrimental effects on various human organs, with a notable concentration of this substance found in the kidneys. This study was to evaluate the impact of intraperitoneal administration of Cu2+ at a dose of 10 mg/kg and investigate the protective effects of Vitamin C, administered at 80 and 100 mg/kg, on the kidneys of male Wistar rats for 10 days, with injections given once a day for one minute. Materials and Methods: Changes in renal histological, stereological, and biochemical parameters were evaluated. Results: The intraperitoneal administration of Cu2+ at 10 mg/kg, unlike Vitamin C (80 and 100 mg/kg), resulted in significant histological and stereological changes in the kidneys of male Wistar rats. These changes included vascular hyperemia, extensive hemorrhage, tissue atrophy, and a reduction in the volume of the left kidney (p < 0.05). Furthermore, exposure to Cu2+ resulted in oxidative stress, as indicated by a reduction in SOD activity and an elevation in MDA levels in the right kidney (p < 0.05). Notably, the higher dose of Vitamin C mitigated the adverse effects of Cu2+ on the histological, stereological, and biochemical parameters compared to the Cu2+-only group (p < 0.05). Conclusion: Our results proposed that co-treatment with Vitamin C may reverse Cu2+-induced alterations and provide a significant protective effect.