<p>Bacterial pathogens such as <i>Salmonella enterica</i> serovar Typhimurium can resist phagocytosis by macrophages. Here we explored the role of bacterial haem biosynthesis in phagocytosis resistance. Using transposon sequencing (Tn-seq) during <i>Salmonella</i> infection of macrophages, we identify a methyltransferase, SirM, that indirectly inhibits phagocytosis of bacteria. Mechanistically, <i>sirM</i> is activated upon interaction with macrophages and methylates HemL, a key enzyme in haem biosynthesis, resulting in upregulation of haem synthesis by <i>Salmonella</i>. <i>Salmonella</i>-derived haem inhibits Cdc42 activation in a Toll-like receptor 4 (TLR4)-dependent manner to inhibit phagocytosis. Moreover, <i>sirM</i> promotes macrophage death by increasing haem synthesis. Experiments in mouse models show that <i>sirM</i> is required for virulence and confers a competitive advantage over intestinal commensal bacteria during infection. We also found that <i>sirM</i> is distributed among enteric pathogens. Collectively, our findings show that bacterial haem promotes evasion of phagocyte responses and pathogenesis to confer an advantage in the host.</p>

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Salmonella-derived haem inhibits macrophage phagocytosis and promotes infection in mice

  • Zuoqiang Wang,
  • Huang Tang,
  • Wanqiu Huang,
  • Chengyue Wang,
  • Yana Chen,
  • Jingchen Yu,
  • Tao Zhou,
  • Bingjie Wen,
  • Jinjing Ni,
  • Danni Wang,
  • Jing Tao,
  • Siqi Zhu,
  • Lin Lyu,
  • Lei Chen,
  • Jun Li,
  • Qihong Kuang,
  • Daojin Yu,
  • Jianping Liu,
  • Lifeng Pan,
  • Mei Zhang,
  • Yue Xu,
  • Guo-Ping Zhao,
  • Jie Lu,
  • Yu-Feng Yao

摘要

Bacterial pathogens such as Salmonella enterica serovar Typhimurium can resist phagocytosis by macrophages. Here we explored the role of bacterial haem biosynthesis in phagocytosis resistance. Using transposon sequencing (Tn-seq) during Salmonella infection of macrophages, we identify a methyltransferase, SirM, that indirectly inhibits phagocytosis of bacteria. Mechanistically, sirM is activated upon interaction with macrophages and methylates HemL, a key enzyme in haem biosynthesis, resulting in upregulation of haem synthesis by Salmonella. Salmonella-derived haem inhibits Cdc42 activation in a Toll-like receptor 4 (TLR4)-dependent manner to inhibit phagocytosis. Moreover, sirM promotes macrophage death by increasing haem synthesis. Experiments in mouse models show that sirM is required for virulence and confers a competitive advantage over intestinal commensal bacteria during infection. We also found that sirM is distributed among enteric pathogens. Collectively, our findings show that bacterial haem promotes evasion of phagocyte responses and pathogenesis to confer an advantage in the host.