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<p>Abscess formation post-surgical incision is a frequent occurrence where a collection of pus forms around the incised region coupled with inflammation. Though patients who develop abscess are often treated by pus drainage and the administration of antibiotics, precise identification of the pathogenic bacteria is critical for effective treatment. Here, we describe the case of a patient, who had developed an abdominal abscess as a result of previous ambulatory peritoneal dialysis. Pus culture coupled with MALDITOF and 16S rRNA sequencing revealed <i>T. tyrosinosolvens</i>, previously misdiagnosed as Tuberculosis, was the main cause of the abscess formation. When treatment was modified from oral rifampicin, isoniazid, ethambutol, and pyrazinamide to intravenous ceftriaxone and oral clarithromycin for 2&#xa0;weeks, and then continuous treatment with oral clarithromycin for 6&#xa0;months, the wound was completely healed with no sign of resurgence. This case study illustrates that the correct identification of a causative organism is essential for administering the right therapy to patients.</p>

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Abdominal Wall Abscess Caused by Tsukamurella tyrosinosolvens Misdiagnosed as Tuberculosis: A Case Report

  • 期刊论文

  • 发表日期：

    2025年6月17日

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  • Yudan Zhang,
  • Zhenghua Wu,
  • Tianyu Zhou,
  • Abdelhak Ouzaouit,
  • Tingya Jiang,
  • Pengfei Zhao,
  • Hua Chen

摘要

Abscess formation post-surgical incision is a frequent occurrence where a collection of pus forms around the incised region coupled with inflammation. Though patients who develop abscess are often treated by pus drainage and the administration of antibiotics, precise identification of the pathogenic bacteria is critical for effective treatment. Here, we describe the case of a patient, who had developed an abdominal abscess as a result of previous ambulatory peritoneal dialysis. Pus culture coupled with MALDITOF and 16S rRNA sequencing revealed T. tyrosinosolvens, previously misdiagnosed as Tuberculosis, was the main cause of the abscess formation. When treatment was modified from oral rifampicin, isoniazid, ethambutol, and pyrazinamide to intravenous ceftriaxone and oral clarithromycin for 2 weeks, and then continuous treatment with oral clarithromycin for 6 months, the wound was completely healed with no sign of resurgence. This case study illustrates that the correct identification of a causative organism is essential for administering the right therapy to patients.