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<p>This report presents new data on the development of oxidative stress (OS) in chronic headache (HA). The main biochemical characteristics of OS are presented. Changes in MRI spectroscopy and biochemical markers confirming the development of OS in migraine are described. The absence of significant differences in measures of OS in migraine with and without aura is emphasized. Pathophysiological differences between migraine and chronic tension headache, in which OS is not detected, are demonstrated. Possible activation of TRPA1 ion channels and calcitonin gene-related peptide in OS, which provokes migraine attacks, is discussed. Directions for drug correction of OS in chronic headache are considered.</p>

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Oxidative Stress in the Pathogenesis of Chronic Headache

  • 期刊论文

  • 发表日期：

    2025年3月1日

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  • A. I. Fedin

摘要

This report presents new data on the development of oxidative stress (OS) in chronic headache (HA). The main biochemical characteristics of OS are presented. Changes in MRI spectroscopy and biochemical markers confirming the development of OS in migraine are described. The absence of significant differences in measures of OS in migraine with and without aura is emphasized. Pathophysiological differences between migraine and chronic tension headache, in which OS is not detected, are demonstrated. Possible activation of TRPA1 ion channels and calcitonin gene-related peptide in OS, which provokes migraine attacks, is discussed. Directions for drug correction of OS in chronic headache are considered.