<p>Whether high-intensity exercise training can effectively mitigate orthostatic-induced post-exercise hypotension (PEH) has not been satisfactory resolved to date. We therefore examined whether 2&#xa0;weeks of high-intensity exercise training can effectively mitigate the orthostatic-induced PEH by conducting comparisons pre- and post-training at the same absolute and relative workloads. Eleven healthy men underwent 2&#xa0;weeks of high-intensity exercise training within the severe intensity domain (i.e., above critical power) for 15&#xa0;min per session for 14 daily consecutive sessions. Assessment of orthostatic-induced PEH was conducted at pre- and post-training (both absolute and relative workloads) across seated and standing recovery postures. The exercise intensities for the PEH trials were conducted at 10% above critical power, followed by a 1-h recovery across seated and standing postures. Post-training, PEH assessment was performed at the same absolute and relative power output (i.e., 10% above the post-intervention critical power) as pre-training. Two weeks of high-intensity exercise training significantly increased <InlineEquation ID="IEq1"> <EquationSource Format="TEX">\(\dot{V}\)</EquationSource> <EquationSource Format="MATHML"><math> <mover accent="true"> <mi>V</mi> <mo>˙</mo> </mover> </math></EquationSource> </InlineEquation>O<sub>2max</sub>, resting ejection fraction, and fractional shortening velocity (all <i>P</i> &lt; 0.01), thereby confirming an increased training status. Consequently, the reduction of systolic, diastolic, and mean arterial blood pressures from baseline were smaller following 2&#xa0;weeks of high-intensity exercise training in both absolute and relative workloads during the recovery across seated and standing postures (all <i>P</i> &lt; 0.01). We conclude that 2&#xa0;weeks of high-intensity exercise training was able to mitigate the orthostatic-induced PEH with both absolute and relative workloads in healthy Asian men.</p>

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High-intensity exercise training mitigates post-exercise orthostatic hypotension in men

  • Tze-Huan Lei,
  • Hao-Yu Li,
  • Richie P. Goulding,
  • Faming Wang,
  • Tatsuro Amano,
  • Tomomi Fujimoto,
  • Naoto Fujii,
  • Narihiko Kondo,
  • Blake Perry,
  • Toby Mündel

摘要

Whether high-intensity exercise training can effectively mitigate orthostatic-induced post-exercise hypotension (PEH) has not been satisfactory resolved to date. We therefore examined whether 2 weeks of high-intensity exercise training can effectively mitigate the orthostatic-induced PEH by conducting comparisons pre- and post-training at the same absolute and relative workloads. Eleven healthy men underwent 2 weeks of high-intensity exercise training within the severe intensity domain (i.e., above critical power) for 15 min per session for 14 daily consecutive sessions. Assessment of orthostatic-induced PEH was conducted at pre- and post-training (both absolute and relative workloads) across seated and standing recovery postures. The exercise intensities for the PEH trials were conducted at 10% above critical power, followed by a 1-h recovery across seated and standing postures. Post-training, PEH assessment was performed at the same absolute and relative power output (i.e., 10% above the post-intervention critical power) as pre-training. Two weeks of high-intensity exercise training significantly increased \(\dot{V}\) V ˙ O2max, resting ejection fraction, and fractional shortening velocity (all P < 0.01), thereby confirming an increased training status. Consequently, the reduction of systolic, diastolic, and mean arterial blood pressures from baseline were smaller following 2 weeks of high-intensity exercise training in both absolute and relative workloads during the recovery across seated and standing postures (all P < 0.01). We conclude that 2 weeks of high-intensity exercise training was able to mitigate the orthostatic-induced PEH with both absolute and relative workloads in healthy Asian men.