Background <p>An anterior cruciate ligament (ACL) rupture causes sensorimotor dysfunction and requires long-term rehabilitation to restore function close to its original level and delay degenerative progression. This dysfunction and rehabilitation can also be seen as a&#xa0;prototype for the deficits associated with deconditioning and aging.</p> Aim of the study <p>The primary intervention, “sensorimotor learning”, will be justified based on the consequences of sensory losses and regenerative processes.</p> Materials and methods <p>The structural and functional disorders, the regenerative processes of somatosensory function, the goals of surgery as well as the goals and contents of active rehabilitation are presented.</p> Results and discussion <p>Early in life, the somatosensory system provides the cerebral body schema. It is the reference for identifying joint positions, the relative positions of body parts, spatial orientation, and body perception during every movement. An ACL rupture destroys sensory sources. The altered afferent and, consequently, central excitation pattern impair muscle activation and movement regulation. Sensory regeneration, which originates from the neural structures of the ACL stumps, is highly heterogeneous between individuals, depending on age and the postoperative period. The deviations from the original sensory array are significant, leaving a&#xa0;deficient information base for the organization and regulation of movement. The surgeon must limit biomechanical instability and establish the basis for neural regeneration. The focus of rehabilitation is sensorimotor learning of explosive strength and speed-oriented loads, with increasing proportions of reactive and unexpected motor demands.</p>

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Ruptur des vorderen Kreuzbands: ein Prototyp gestörter Sensomotorik und Rehabilitation

  • W. Laube,
  • E. Kurz,
  • R. Schwesig,
  • T. Bartels

摘要

Background

An anterior cruciate ligament (ACL) rupture causes sensorimotor dysfunction and requires long-term rehabilitation to restore function close to its original level and delay degenerative progression. This dysfunction and rehabilitation can also be seen as a prototype for the deficits associated with deconditioning and aging.

Aim of the study

The primary intervention, “sensorimotor learning”, will be justified based on the consequences of sensory losses and regenerative processes.

Materials and methods

The structural and functional disorders, the regenerative processes of somatosensory function, the goals of surgery as well as the goals and contents of active rehabilitation are presented.

Results and discussion

Early in life, the somatosensory system provides the cerebral body schema. It is the reference for identifying joint positions, the relative positions of body parts, spatial orientation, and body perception during every movement. An ACL rupture destroys sensory sources. The altered afferent and, consequently, central excitation pattern impair muscle activation and movement regulation. Sensory regeneration, which originates from the neural structures of the ACL stumps, is highly heterogeneous between individuals, depending on age and the postoperative period. The deviations from the original sensory array are significant, leaving a deficient information base for the organization and regulation of movement. The surgeon must limit biomechanical instability and establish the basis for neural regeneration. The focus of rehabilitation is sensorimotor learning of explosive strength and speed-oriented loads, with increasing proportions of reactive and unexpected motor demands.